Anandakuttan biography templates
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Beomjune Shin
1George W. Woodruff School of Mechanical Engineering, Georgia Institute of Technology, Atlanta, GA, , USA
2Wearable Intelligent Systems and Healthcare Center (WISH Center), Institute for Matter and Systems, Georgia Institute of Technology, Atlanta, GA, , USA
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Sung Hoon Lee
2Wearable Intelligent Systems and Healthcare Center (WISH Center), Institute for Matter and Systems, Georgia Institute of Technology, Atlanta, GA, , USA
3School of Electrical and Computer Engineering, Georgia Institute of Technology, Atlanta, GA, , USA
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Kangkyu Kwon
2Wearable Intelligent Systems and Healthcare Center (WISH Center), Institute for Matter and Systems, Georgia Institute of Technology, Atlanta, GA, , USA
3School of Electrical and Computer Engineering, Georgia Institute of Technology, Atlanta, GA, , USA
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Milestones
Foundation was registered under Charitable Societies Registration Act.
Release of the book 'Priya Smaranakal'.
'Souvenir' released.
Release of the book 'N. Krishna Pillayude Lekhunatakangal'.
Release of the book 'Ithilkkanniyum Koonamkurukkum'.
Unveiling the Portrait of N. Krishna Pillai at Thiruvananthapuram Public Library.
Release of the book 'Adiverukal'.
Drama Festival,
Felicitation of Dramatists,
Release of the book 'Kalidasan Muthal O.N.V. Vare'.
Drama Festival,
'Bhagnabhavanam' - Golden Jubilee.
Kalotsavam ,
Release of 'N. Krishna Pillai' - Biography
Kalotsavam ,
Release of the book 'Irullum Velichavum'.
Kalotsavam
Kalotsavam
Kalotsavam
O.N.V. Felicitation.
Kalotsavam
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Substrate Specific Inhibitor Designed against the Immunomodulator GMF-beta Reversed the Experimental Autoimmune Encephalomyelitis
Introduction
Multiple sclerosis is a chronic autoimmune, demyelinating, neurodegenerative disorder of the central nervous struktur (CNS) affecting million people globally1,2. Despite different disease-modifying therapies adopted to mitigate the inflammatory milieu of MS using different drugs3,4,5, patient’s progress from an acute phase to a stage with considerable neurological disabilities. Current therapies modulate disease differently as they target activated T cells, antigen presenting cells or prevent the egress of T cells into brain. Importantly, none of these drugs control molecules that modulera the proinflammatory response at a fundamental level following infiltration of activated T and B lymphocytes6,7. Activated lymphocytes undergo clonal expansion assisted by the cytokines produced mainly by astrocytes and microglia and glia maturation